Kimberly Eyer

This study links the subgingival microbiome in rheumatoid arthritis to elevated CPAs via enrichment of A. butyrica, P. simiae, and P. stomatis. These bacteria contribute to citrullination and may be key microbial biomarkers or BTI targets in A-associated periodontitis.

This study found that oral microbiota in early rheumatoid arthritis displays distinct bacterial enrichments and metabolic shifts. Key taxa, including Filifactor alocis and Porphyromonas endodontalis, were elevated in A regardless of periodontal status, highlighting their potential as biomarkers and microbial contributors to A pathogenesis.

This study links R38 genotype to oral microbiota composition in rheumatoid arthritis, revealing microbial shifts modulated by taste receptor polymorphisms. Findings suggest buccal dysbiosis in A is partially shaped by host genetics, offering new insight into mucosal immunity and potential biomarkers for disease risk and microbial-targeted therapy.

This study used gut microbiome metagenomics in rheumatoid arthritis to identify Prevotella enrichment and redox gene loss in A. Findings reveal microbiome-host genomic interactions and metabolic shifts that define A pathogenesis, offering population-specific microbial signatures and potential therapeutic targets based on microbial function and immunogenetic alignment.

This study on the subgingival microbiome in rheumatoid arthritis and periodontitis identified Treponema enrichment in A. Despite similar microbial richness across groups, specific dysbiotic shifts suggest that oral bacteria, particularly Treponema and Porphyromonas, may bridge periodontal and systemic inflammation in A pathogenesis.

This study on intestinal microbiota in rheumatoid arthritis spouses reveals shared dysbiosis, with enrichment of Streptococcus and Blautia and depletion of Roseburia. These shifts implicate environment-driven microbial patterns in A pathogenesis and highlight potential microbiome-based interventions for at-risk populations.

This study explores the gut microbiome in early rheumatoid arthritis, revealing Prevotella enrichment and Collinsella depletion. These microbial shifts, seen before full disease onset, highlight the gut microbiome in early rheumatoid arthritis as a potential therapeutic target and biomarker for disease progression.

This review highlights how oral microbiota in rheumatoid arthritis patients shows pathogenic enrichment, increased cytokines, and worsened periodontal outcomes. These dysbiotic patterns, involving taxa like Prevotella and Parvimonas, suggest diagnostic and therapeutic implications, reinforcing the need to monitor oral health in A.

This study reveals oral microbiota dysbiosis in rheumatoid arthritis, with distinct shifts in high-risk individuals before clinical symptoms. Prevotella_6 and Rothia were enriched, while Neisseria oralis was depleted, suggesting early microbial signatures and potential targets for diagnostic and preventive strategies in rheumatoid arthritis.

Subgingival microbiota in rheumatoid arthritis patients shows citrulline-producing and pro-inflammatory species enrichment despite periodontal health. These findings highlight Cryptobacterium curtum and networked anaerobes as potential contributors to A pathogenesis and offer insight into early microbial biomarkers for immune-mediated disease.

Subgingival microbiome in rheumatoid arthritis reveals microbial community shifts driven by disease activity, smoking, and treatment. Prednisolone use correlates with healthier profiles, while smoking promotes dysbiosis, highlighting microbial biomarkers for A management.

This study identifies oral microbiome biomarkers for arthritis screening, distinguishing A from A using salivary microbial profiles. It highlights Prevotella and Veillonella in A and Rothia and Streptococcus in A, supporting microbiome-based diagnostics.

Rheumatoid arthritis gut microbiome dysbiosis is marked by Collinsella expansion and Faecalibacterium loss, disrupting gut barrier function and promoting inflammation. These findings reveal potential diagnostic biomarkers and mechanistic pathways for microbiome-targeted interventions.

This study shows that the oral microbiota in early rheumatoid arthritis includes enriched Tannerella forsythia and Streptococcus anginosus, correlating with increased periodontal attachment loss. These findings suggest early microbial dysbiosis may influence A pathogenesis and serve as diagnostic biomarkers or targets for preventive care.

Oral microbiota in new-onset rheumatoid arthritis shows unique enrichment of Prevotella, Leptotrichia, and Anaeroglobus, independent of periodontitis severity. These taxa may act as microbial triggers or biomarkers for autoimmunity, suggesting the oral microbiome’s relevance in A onset and potential for diagnostic or therapeutic targeting.